Definition
- Hyperthyroidism occurs when there is accelerated thyroid hormone biosynthesis and secretion by the thyroid gland.
- Thyrotoxicosis is the hypermetabolic clinical syndrome resulting from serum elevations in thyroid hormone levels, specifically free thyroxine (FT4) and/or triiodothyronine (FT3).
- Thyroid storm is the extreme manifestation of thyrotoxicosis that is life-threatening and a medical emergency, often precipitated by surgery, trauma, or infection.
Etiologies
- Graves’ disease (diffuse toxic goiter): most common cause. Diffuse enlargement of thyroid, ± exophthalmos, ± dermopathy (pretibial myxedema) with TSH-receptor auto-antibodies (TRAb). Can be associated with other autoimmune disorders (e.g. T1DM, pernicious anemia). Risk factors are not well-defined but may include genetic factors, high iodine intake, stress, smoking, and the postpartum state.
- Toxic adenoma/toxic multinodular goiter: hyperplasia of thyroid follicular cells due to activating mutations; independent of TSH. Sometimes associated with low iodine intake.
- Subacute thyroiditis: often due to viral infection. Thyrotoxicosis due to release of stored thyroid hormone, followed by euthyroid and/or hypothyroid phases, then recovery. May be painful (de Quervain’s) or painless (subacute lymphocytic thyroiditis, post-partum thyroiditis).
- Other causes: iodine load-induced (the Jod-Basedow phenomenon), exogenous ingestion (thyrotoxicosis factitia), struma ovarii (thyroid tissue in ovarian tumor), pituitary TSH-secreting adenoma, early Hashimoto’s (hashi-toxicosis), amiodarone-induced, trophoblastic tumors (hCG stimulation of TSH receptor).
Evaluation
Symptoms and signs
- Signs of hypermetabolic state: restlessness, tachycardia, diaphoresis, hyper-defecation, weight loss, palpitations. Can also see lid lag, heat intolerance, tremor, hyperreflexia, moist skin and fine hair.
- Apathetic hyperthyroidism: elderly patients often don’t have classic symptoms, but may present with lethargy, weight loss, shortness of breath and atrial fibrillation.
Associated conditions/complications
- Heart failure, osteoporosis, hypercalcemia, nephrolithiasis. May account for 10-15% of new cases of atrial fibrillation.
Thyroid storm (severe thyrotoxicosis)
- Fever (often very high 104-106ºF), delirium, stupor, coma, diaphoresis, tachycardia, arrhythmias, CHF, vomiting/diarrhea, hepatic failure.
- Often induced by illness, surgery, or iodine administration, can also be seen with long untreated hyperthyroidism.
Work-up
- TSH suppressed (except in pituitary disease), FT4 and/or FT3 increased.
- ESR can be increased in subacute thyroiditis.
- Anti-thyroperoxidase antibodies often positive in silent thyroiditis.
- TSH receptor Ab (TRAb) positive in Graves’ disease.
- Radioactive iodine uptake can be useful to differentiate between causes (contraindicated in pregnant patients): diffuse high uptake in Graves’, focal high uptake with toxic nodule(s), very low uptake in subacute thyroiditis.
- Burch-Wartofsky Point Scale (BWPS) is a scoring system to help identify thyroid storm.
Management
Medical therapy
- Up to 50% of patients with Graves’ disease can go into spontaneous remission.
- Symptom management: propranolol to control symptoms and tachycardia (blocks adrenergic effects and peripheral conversion of T4 to T3). Beta blockade can be stopped once anti-thyroid treatment takes effect.
- Inhibition of hormone synthesis: methimazole or propylthiouracil (PTU). May be used as short-term management until definitive therapy, or for long-term use. Methimazole is generally preferred due to better side effect profile (PTU preferred in pregnancy). Risk of agranulocytosis, seen in 0.5%, usually early in treatment, also risk for hepatic toxicity. Obtain baseline CBC and LFTs.
Definitive therapy
- Radioactive iodine ablation with I-131 or surgery, generally rendering patient hypothyroid and requiring thyroid hormone replacement.
Thyroid storm
- Consult Endocrinology as mortality is 10-20%.
- Supportive therapy with IVF, O2, and acetaminophen, likely ICU.
- Careful search for precipitating causes (e.g. infection). Avoid ASA, as it can displace FT4 from TBG.
- Immediately start PTU 200-300 mg PO q 4-6 hours.
- Follow with saturated solution of potassium iodide (SSKI) to inhibit conversion of T4 to T3.
- Consider propranolol 60-80 mg PO q 4-6 hours to control tachycardia (caution in heart failure).
- Consider hydrocortisone 50-100 mg IV q 6-8 hours x 48 hours (blocks conversion of T4 to T3).
Key Points
- Most hyperthyroidism is caused by Graves’ disease.
- Hyperthyroidism can manifest as atrial fibrillation, CHF, osteoporosis, or weight loss/fatigue in elderly.
- Treatment consists of propanolol, thionamides, radioactive ablation, or surgery.
- Thyroid storm is a clinical emergency and prompt diagnosis and treatment are essential.
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Ross DS, Burch HB, Cooper DS, et al. 2016 American Thyroid Association Guidelines for Diagnosis and Management of Hyperthyroidism and Other Causes of
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